NutriCoach brings the full Henselmans Personal Trainer Certification curriculum to your fingertips — personalised to your body, your goals, and your training. Work with a certified coach, track everything, and actually understand why.
Your personalised coaching journey — work through each stage to unlock new tools and insights.
| Fat Type | Sources | Anabolic Mechanism | Cap |
|---|---|---|---|
| PUFA — omega-3 | Fatty fish, fish oil | Direct MPS stimulation, anti-inflammatory, lowers cortisol, raises testosterone | Target 2g EPA+DHA/d |
| PUFA — omega-6 | Sunflower oil, nuts, seeds | Anabolic (LIPOGAIN data: 3× more LBM vs SFA overfeeding), not pro-inflammatory in whole foods | No restriction needed |
| MUFA | Olive oil, avocado, eggs, almonds | Anti-inflammatory, improves cholesterol profile, likely anabolic (olive/oleuropein data) | No specific cap |
| SFA | Dairy, meat, eggs, coconut | Indirect: SFA → cholesterol precursor → testosterone/steroid hormones. No direct MPS effect. | ≤33% of total fat |
| Trans fat (industrial) | Margarine, fried fast food | No benefit. Atherogenic, pro-inflammatory. Avoid entirely. | 0 — avoid |
| Activity Type | Keto Impact | Mechanism | Evidence |
|---|---|---|---|
| Strength / Powerlifting | ✅ No impact | Glycogen depletion from strength training is modest; Cori cycle recycles lactate → glucose; resynthesis complete within 24h | Multiple RCTs (Paoli 2012, Wilson 2017, Sawyer 2013, Paoli 2021) |
| Bodybuilding / Hypertrophy | ✅ No impact | Same as strength — glycogen rarely depleted >39% even at high training volume | Paoli 2021 (competitive natural bodybuilders); Meirelles & Gomes 2016 |
| CrossFit / HIIT | ✅ Generally no impact | Performance maintained; mild acidosis may limit peak anaerobic output transiently | Gregory 2016; even 0.6 g/kg/d carbs preserved CrossFit performance |
| Low-Intensity Endurance | ✅ No impact or beneficial | At low intensity, fat replaces glycogen as primary fuel — no performance loss | Consistent across multiple endurance studies |
| High-Intensity Endurance | ⚠️ Anaerobic endurance impaired | Mild ketosis-induced acidosis may limit rate of anaerobic metabolism; Wingate work impaired (Zajac et al.) | Most affected: team sports, sprint intervals, >90% VO2max efforts |
| Team Sports | ❌ Impaired | High-volume anaerobic demands exceed what fat oxidation + Cori cycle can sustain | Avoid strict keto for sport athletes with repeated sprint demands |
Answer 8 quick questions about your diet to identify your highest-risk micronutrient gaps.
| Nutrient | RDA / AI (♀/♂) | Upper Limit | Key Role for Athletes | Best Food Sources | Supplement Form |
|---|
Based on your screener flags, generate a tailored supplement protocol with form, dose and timing guidance.
Select a mineral to see what helps or blocks its absorption. Critical for supplement timing.
Check if a multivitamin passes Henselmans' quality criteria before recommending it.
The 7 micronutrients most commonly deficient in athletes. Food sources that are semi-essential + supplementation strategy if diet falls short.
| Micronutrient | Why Athletes Need It | Semi-essential food sources | Supplementation strategy |
|---|---|---|---|
| Vitamin D3 | Strength, LBM, testosterone, injury resistance, immune function | Sun exposure (10–30 min/day, near-full body, UV index 3+) | 2,000–9,000 IU/d Vitamin D3 (not D2). Take with fat. Target serum 25(OH)D ~115 nmol/L. Bloodwork advised. |
| Magnesium | Testosterone, strength, sleep quality, stress response, insulin sensitivity | Spinach, mackerel, fermented/soaked whole grains, nuts, dark chocolate, potato | Up to 6 mg/kg/d Citrate, chloride, lactate or aspartate — NOT oxide or glycinate. Avoid taking with calcium. |
| Vitamin K | Blood clotting, bone formation (synergy with D3 + calcium). K2 linked to reduced all-cause mortality. | Leafy greens (K1), aged cheese/natto (K2), avocado, pumpkin | 1+ mcg/kg/d Phylloquinone (K1) & menaquinone MK-7 (K2). Take with a fatty meal. |
| Calcium | Bone health, neuromuscular function, satiety signalling, fat partitioning in athletes | Dairy (best), sardines with bones, tofu, cruciferous veg in large amounts | 800 mg/d Calcium citrate preferred. Avoid with magnesium or zinc. Max ~800 mg absorbed per serving. |
| Iodine | Thyroid hormone synthesis — directly controls metabolic rate | Iodized salt (NOT sea salt), dairy (2 cups milk/yogurt), marine seafood, seaweed weekly | 150+ mcg/d Standardized kelp extract. Sea salt does not count — myth. |
| Zinc | Testosterone, protein synthesis, immune function, wound healing. Doubling possible from correcting mild deficiency. | Red meat, shellfish (oysters = 1/day covers it), dark chicken meat | 10–15 mg/d Any form. Avoid with dairy or calcium-rich meal. Max 40 mg/d (copper depletion risk above). |
| Iron | O₂ transport, endurance, resistance to fatigue, strength. 52% of female athletes deficient. | Shellfish, sardines, liver/kidney (1–3×/wk for female athletes), beef daily | ⚠ Risky Bloodwork FIRST. Toxicity risk for men. Ferrous salts or heme polypeptides if needed. UL = 45 mg/d. |
Type any food to instantly see its place in Henselmans' 6-tier health hierarchy with rationale. Based on the PTC Week 11 food quality framework.
Complete 6-tier reference. Note: tiers reflect health outcomes — not anabolic potential or satiety index, which are separate considerations.
Strength training impairs performance from ~2.5% dehydration. Overhydration (hyponatremia) is a real risk too. Find your personal optimum.
Up to 20% of the Western population has GI distress. 70–75% of IBS patients respond to a low-FODMAP protocol. Identify your suspect categories and get a structured elimination plan.
Search any food to check its FODMAP status. Portion sizes matter — some foods are safe in small amounts only. Source: IBSDiets + Monash University guidelines.
| Food | Category | FODMAP Status | Portion / Notes |
|---|
Build up to 4 meal components and see the weighted satiety index vs calories. Reference: white bread = 100% SI (Holt et al. 1995).
Identify the behavioural and physiological factors currently driving your appetite above its baseline, with ranked priorities for fixing them.
Tell us your current food choices and get science-backed satiety-maximising swaps with estimated calorie reduction.
From Henselmans PTC Week 12 — Hunger Management client guide. Click any strategy to expand the science behind it.
Diet breaks restore hormonal adaptations (leptin, T3, cortisol), reduce psychological fatigue, and may preserve lean mass better than uninterrupted dieting. Henselmans recommends 1–2 week breaks at maintenance every 8–12 weeks of dieting.
Reverse dieting incrementally increases calories post-cut to restore metabolic rate while minimising fat regain. The rate of increase depends on client psychology, fat gain tolerance, and how deep the deficit was.
Estimate total adaptive thermogenesis based on diet depth and duration. Informs reverse diet endpoint and maintenance calorie expectations.
Based on current body composition and goals, get a recommended multi-phase sequence for the next 12 months. [HL PTC Week 13]
Key communication and behaviour-change frameworks from PTC Week 13. Click to expand each domain.
Identify your acne type and severity based on symptom description. The first step in treatment is differential diagnosis — the right treatment depends entirely on lesion type.
Step-by-step treatment escalation following Henselmans PTC recommendations. Always begin with the lowest-risk interventions.
Log dietary changes and track acne flares over time. Insulin/IGF-1 signalling is the primary dietary pathway for acne. Key triggers: dairy (esp. skim milk), high-GI carbohydrates, processed foods, chocolate.
Side-by-side comparison of all major topical acne and PIH treatments from the PTC module. Click any row to expand clinical notes.
| Treatment | Use case | Evidence | Rx needed? | Key caution |
|---|
Post-inflammatory hyperpigmentation treatment protocol, ordered by evidence strength. Daily SPF 15+ is non-negotiable — sunburn greatly worsens PIH.
Key differentiators to guide correct treatment approach. Misdiagnosis = ineffective treatment.
Identify severity grade and understand the structural cause. Cellulite is graded 0–4 based on visual appearance and skin texture.
The anti-cellulite industry is enormous and most research is industry-sponsored and methodologically weak. This table rates treatments by independent high-quality RCT evidence. Click any row to expand notes. [HL Zerini et al. 2015]
| Treatment | Evidence quality | Effectiveness | Durability | Verdict |
|---|
Estimate how much fat loss is needed to significantly reduce cellulite visibility. Based on the correlation between body fat % and cellulite severity from PTC.
Sex differences in subcutaneous tissue architecture explain why 85–98% of women have cellulite vs. a small minority of men at the same body fat level. [HL Nurnberger & Muller 1978]
Generate a ready-to-use, evidence-based explanation for clients asking about cellulite treatments. Customise by product type the client is considering.
Calculates carbload carbohydrate targets based on bodyweight, depletion level, sex, and division. Carbload should be at least 8 g/kg, preferably 10 g/kg. Women need energy surplus to respond. [HL Sherman 1981, James et al. 2001]
Generate a personalised hour-by-hour peak week countdown from show time. Protocol varies significantly by division and individual. Always trial-run before competition.
Build a personalised pre-stage vascularity meal. The "shitload" maximises insulin-driven vasodilation. Individual response varies enormously — always trial-run in pre-workout meals.
Based on physique attributes and goals, find the best-fit competition division. Based on coach ratings from Fagerli, Norton, Wilson & Henselmans (PTC survey).
Monitor these signs during water cut trials. Stop dehydration and sip water + minerals if any red-flag symptoms appear. Posing > dryness.
Pump up just minutes before stage. 12–30 reps, short rest, controlled eccentric / explosive concentric, mid-range only. Pump = +20% cross-section for 5 min; gone within 30–60 min.
Measure in the morning before eating. Relax the muscle being measured. Record both sides where applicable.
| Site | L / Single | R |
|---|---|---|
Bicep Flexed mid-upper arm | ||
Chest Nipple line, exhale | — | |
Waist (2″ above) 2 inches above belly button | — | |
Waist (navel) At belly button — key metric | — | |
Waist (2″ below) 2 inches below belly button | — | |
Hip Widest point | — | |
Thigh Mid-thigh, relaxed | ||
Calf Widest point, standing |
When you submit your weekly check-in, the adjustment engine will automatically account for your cycle phase. Weight increases during the late luteal and menstrual phases will be flagged as likely water retention — not fat gain — so macros won't be incorrectly reduced.
What do you want to achieve in the next 8–16 weeks?
Where do you ultimately want to be? The planner will map the full phase sequence to get you there.
Distinguish simple dandruff from seborrheic dermatitis and receive a personalised treatment protocol. Based on Borda & Wikramanayake (2015) classification criteria and Henselmans PTC treatment hierarchy.
Not all active ingredients suit every person. Hair colour, beard goals, and prior shampoo history all affect optimal choice. Top 3 ingredients ranked by evidence and compatibility. [Borda & Wikramanayake 2015]
Factors that exacerbate dandruff and seborrheic dermatitis. Explains winter prevalence and why stress worsens skin conditions. [HL PTC]
Comparison adapted from Borda & Wikramanayake (2015). Both share a fungal root cause but differ significantly in presentation and treatment intensity.
"Beard itch" is most often seborrheic dermatitis of the beard area — essentially facial dandruff. Requires specific ingredient selection because some antifungals have anti-androgenic effects that can reduce beard fullness with prolonged use.
Distinguish between the 3 main hair loss types based on pattern, onset, and pull test. Each has a different cause and treatment approach. Based on Henselmans PTC differential diagnosis algorithm.
Personalised step-by-step treatment protocol for androgenetic alopecia based on sex, severity, beard goals, and risk tolerance. None offer a cure — all slow progression. [Henselmans PTC 2021]
Telogen effluvium is stress-triggered shedding. Onset is typically 2–3 months after the trigger. Treatment = address root cause. [HL PTC]
Evidence-ranked comparison of all major treatments. No true cure exists — all slow progression. [Henselmans PTC 2021]
Evidence-based dosing reference for the two most established pharmacological AGA treatments. Read all safety notes before use. Consult a physician before starting any DHT-blocking therapy.
Calculates your optimal daily sun exposure range based on skin type, goal, location, and time of day. Sweet spot = ~50% of sunburn threshold. Based on Henselmans PTC tanning module and Fitzpatrick scale.
Most people apply sunscreen at ~25% of the required 2mg/cm² density. Real-world protection = ⁴√(label SPF) at normal application. This tool shows your actual protection level. [Henselmans PTC 2021]
Understanding which UV type does what determines when and how to tan safely. [HL PTC]
Ranked by safety, tanning effect, vitamin D production, and practical considerations. [HL PTC]
Sun-derived vitamin D is far more effective than supplementation — equivalent exposure raises serum levels 4× more than 1,000 IU D3 capsule. No toxicity risk from sun (production self-regulates). Protect face — it contributes little vitamin D but has highest damage + cancer sensitivity. [Holick 2008]
The hypothalamic-pituitary-gonadal axis governs sex hormone production in both sexes. AAS suppress this cascade via negative feedback. [HL PTC — exam-relevant]
Mayo Clinic / 120-lab US survey reference ranges for monitoring hormone status pre-, on-, and post-cycle. Free testosterone is the primary diagnostic marker for men. [HL PTC]
Enter bloodwork values to assess hormone status and flag out-of-range markers. Reference ranges from Mayo Clinic / HL PTC. For men: free testosterone is the key post-cycle recovery indicator.
hCG (LH analog) maintains testicular size, fertility, and function during AAS suppression. Used on-cycle only — NOT during PCT. [HL PTC]
SERMs block estrogen receptors at the hypothalamus, lifting negative feedback and restoring LH/FSH → testosterone production. Men only — SERMs not effective in women. [HL PTC]
SARMs (Selective Androgen Receptor Modulators) are marketed as safer AAS alternatives. The evidence says otherwise. Summary of Dalton et al. (2011) — ostarine/MK-2866 — and Basaria et al. (2013) — LGD-4033. [HL PTC]
Evidence-based ratings for the most commonly used sports supplements, based on Henselmans PTC 2021 and ISSN Position Stands. Click any supplement for detailed notes on mechanism, dosing, and caveats.
The single most evidence-backed ergogenic supplement available. Increases phosphocreatine stores → faster ATP resynthesis → more reps / power output → greater training stimulus → more muscle. [HL PTC]
Caffeine is the second most evidence-supported ergogenic. Builds personalised dosing and timing protocol based on bodyweight and tolerance. [HL PTC]
Select a client goal and training phase to see the evidence-based supplement priority list — ranked from highest to lowest ROI. Only supplements with at least moderate evidence are included. [HL PTC]
The supplement industry is a multi-billion dollar market built largely on weak evidence and marketing. Here are the most pervasive myths debunked with PTC science. [HL PTC]
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